Periostin对心肌梗死后中性粒细胞浸润及心肌修复的调控作用研究(4)
[7]Kalogeris T，Baines CP，Krenz M，Korthuis RJ.Cell biology ofischemia/reperfusion injury.Int Rev Cell Mol Biol，2012，298：229-317.
[8]Mezzaroma E，Toldo S，Farkas D，Seropian IM，Van TassellBW，Salloum FN，Kannan HR，Menna AC，Voelkel NF，Abbate A.The inflammasome promotes adverse cardiac remodel-ing following acute myocardial infarction in the mouse.Proc NatlAcad Sci U S A，2011，108(49)：19725-19730.
[9]Hansson GK.Inflammation and atherosclerosis：the end of a con-troversy.Circulation，2017，136(20)：1875-1877.
, 百拇医药
[10]Izuhara K，Nunomura S，Nanri Y，Ogawa M，Ono J，MitamuraY，Yoshihara T.Periostin in inflammation and allerg3.Cell MolLife Sci，2017，74(23)：4293-4303.
[11]Kudo A，Kii 1.Periostin function in communication with extrace-llular matrices.J Cell Common Signal，2018，12(1)：301-308.
[12]Sasaki H，Dai M，Auclair D，Kaji M，Fukai I，Kiriyama M，Yamakawa Y，Fujii Y，Chen LB.Serum level of the periostin，a homologue of an insect cell adhesion molecule，in thymomapatients.Cancer Lett，2001，172(1)：37-42.
, 百拇医药
[13]Frangogiannis NG.The inflammatory response in myocardialinjury，repair，and remodelling.Nature reviews.Nat RevCardiol，2014，11(5)：255-265.
[14]Faxon DP，Gibbons RJ，Chronos NA，Gurbel PA，SheehanF;HALT-MI Investigators.The effect of blockade of the CD11/CD 18 integrin receptor on infarct size in patients with acutemyocardial infarction treated with direct angioplasty：the resultsof the HALT-MI study.J Am Coll Cardiol，2002，40(7)：1199-1204.
, http://www.100md.com
[15]Wang J.Neutrophils in tissue injury and repair.Cell Tissue Res，2018，371(3)：531-539.
[16]Horckmans M，Ring L，Duchene J，Santovito D，Schloss MJ，Drechsler M，Weber C，Soehnlein 0，Steffens S.Neutrophilsorchestrate post-myocardial infarction healing by polarizingmacrophages towards a reparative phenotype.Ear Heart J，2017，38(3)：187-197.
[17]Shapira I，Rotstein R，Fusman R，Gluzman B，Roth A，KerenG，Avitzour D，Arber N，Berliner S.Combined leukocyte anderythrocyte aggregation in patients with acute myocardial infar-ction.Int J Cardiol，2001，78(3)：299-305.
, 百拇医药
[18]Anzai A，Choi JL，He S，Fenn AM，Nairz M，Rattik S，McAlpine CS，Mindur JE，Chan CT，Iwamoto Y，Tricot B，Wojtkiewicz GR，Weissleder R，Libby P，Nahrendorf M，StoneJR，Becher B，Swirski FK.The infareted myocardium solicitsGM-CSF for the detrimental oversupply of inflammatory leuk-ocytes.J Exp Med，2017，214(11)：3293-3310.
[19]Vinten-Johansen J，Jiang R，Reeves JG，Mykytenko J，DeneveJ，Jobe IJ.Inflammation，proinflammatory mediators and myoca-rdial ischemia-reperfusion Injury.Hematol Oncol Clin NorthAm，2007，21(1)： 123-145., 百拇医药(祝双华 廖延 姜美花 詹宇婷 彭朝权)
[8]Mezzaroma E，Toldo S，Farkas D，Seropian IM，Van TassellBW，Salloum FN，Kannan HR，Menna AC，Voelkel NF，Abbate A.The inflammasome promotes adverse cardiac remodel-ing following acute myocardial infarction in the mouse.Proc NatlAcad Sci U S A，2011，108(49)：19725-19730.
[9]Hansson GK.Inflammation and atherosclerosis：the end of a con-troversy.Circulation，2017，136(20)：1875-1877.
, 百拇医药
[10]Izuhara K，Nunomura S，Nanri Y，Ogawa M，Ono J，MitamuraY，Yoshihara T.Periostin in inflammation and allerg3.Cell MolLife Sci，2017，74(23)：4293-4303.
[11]Kudo A，Kii 1.Periostin function in communication with extrace-llular matrices.J Cell Common Signal，2018，12(1)：301-308.
[12]Sasaki H，Dai M，Auclair D，Kaji M，Fukai I，Kiriyama M，Yamakawa Y，Fujii Y，Chen LB.Serum level of the periostin，a homologue of an insect cell adhesion molecule，in thymomapatients.Cancer Lett，2001，172(1)：37-42.
, 百拇医药
[13]Frangogiannis NG.The inflammatory response in myocardialinjury，repair，and remodelling.Nature reviews.Nat RevCardiol，2014，11(5)：255-265.
[14]Faxon DP，Gibbons RJ，Chronos NA，Gurbel PA，SheehanF;HALT-MI Investigators.The effect of blockade of the CD11/CD 18 integrin receptor on infarct size in patients with acutemyocardial infarction treated with direct angioplasty：the resultsof the HALT-MI study.J Am Coll Cardiol，2002，40(7)：1199-1204.
, http://www.100md.com
[15]Wang J.Neutrophils in tissue injury and repair.Cell Tissue Res，2018，371(3)：531-539.
[16]Horckmans M，Ring L，Duchene J，Santovito D，Schloss MJ，Drechsler M，Weber C，Soehnlein 0，Steffens S.Neutrophilsorchestrate post-myocardial infarction healing by polarizingmacrophages towards a reparative phenotype.Ear Heart J，2017，38(3)：187-197.
[17]Shapira I，Rotstein R，Fusman R，Gluzman B，Roth A，KerenG，Avitzour D，Arber N，Berliner S.Combined leukocyte anderythrocyte aggregation in patients with acute myocardial infar-ction.Int J Cardiol，2001，78(3)：299-305.
, 百拇医药
[18]Anzai A，Choi JL，He S，Fenn AM，Nairz M，Rattik S，McAlpine CS，Mindur JE，Chan CT，Iwamoto Y，Tricot B，Wojtkiewicz GR，Weissleder R，Libby P，Nahrendorf M，StoneJR，Becher B，Swirski FK.The infareted myocardium solicitsGM-CSF for the detrimental oversupply of inflammatory leuk-ocytes.J Exp Med，2017，214(11)：3293-3310.
[19]Vinten-Johansen J，Jiang R，Reeves JG，Mykytenko J，DeneveJ，Jobe IJ.Inflammation，proinflammatory mediators and myoca-rdial ischemia-reperfusion Injury.Hematol Oncol Clin NorthAm，2007，21(1)： 123-145., 百拇医药(祝双华 廖延 姜美花 詹宇婷 彭朝权)