能量代谢在癫痫疾病中的研究进展(4)
[12] Choy M， Wells JA， Thomas DL， Gadian DG， Scott RC， Lythgoe MF. Cerebral blood flow changes during pilocarpine-induced status epilepticus activity in the rat hippocampus. Exp Neurol，2010， 225(1)：196-201.
[13] van Eijsden P， Notenboom RG， Wu O， de Graan PN， van Nieuwenhuizen O， Nicolay K， Braun KP. In vivo 1H magnetic resonance spectroscopy， T2-weighted and diffusion-weighted MRI during lithium-pilocarpine-induced status epilepticus in the rat. Brain Res，2004， 1030(1)：11-18.
, http://www.100md.com
[14] Zsurka G， Kunz WS. Mitochondrial dysfunction and seizures： the neuronal energy crisis. Lancet Neurol，2015， 14(9)：956-966.
[15] Sada N， Lee S， Katsu T， Otsuki T， Inoue T. Epilepsy treatment. Targeting LDH enzymes with a stiripentol analog to treat epi-lepsy. Science，2015， 347(6228)：1362-1367.
[16] 翟瓊香，桂娟，乔惠宪，刘纪清. 小儿癫痫治疗前后脑细胞葡萄糖代谢改变及临床价值. 中山大学学报(医学科学版)，2008，29(2)：198-201.
[17] Shao LR， Stafstrom CE. Glycolytic inhibition by 2-deoxy-d-glu-cose abolishes both neuronal and network bursts in an in vitro seizure model. J Neurophysiol，2017， 118(1)：103-113.
, 百拇医药
[18] Oz G， DiNuzzo M， Kumar A， Moheet A， Seaquist ER. Revisiting glycogen content in the human brain. Neurochem Res，2015， 40(12)：2473-2481.
[19] Walls AB， Sickmann HM， Brown A， Bouman SD， Ransom B， Schousboe A， Waagepetersen HS. Characterization of 1，4-dideoxy-1，4-imino-d-arabinitol (DAB) as an inhibitor of brain glycogen shunt activity. J Neurochem，2008， 105(4)：1462-1470.
[20] DiNuzzo M， Mangia S， Maraviglia B， Giove F. Does abnormal glycogen structure contribute to increased susceptibility to seiz-ures in epilepsy？ Metab Brain Dis，2015， 30(1)：307-316.
, http://www.100md.com
[21] Gibbs ME， Lloyd HG， Santa T， Hertz L. Glycogen is a preferred glutamate precursor during learning in 1-day-old chick： biochemical and behavioral evidence. J Neurosci Res，2007， 85(15)：3326-3333.
[22] Johnson J Jr， Pajarillo E， Karki P， Kim J， Son DS， Aschner M， Lee E. Valproic acid attenuates manganese-induced reduction in expression of GLT-1 and GLAST with concomitant changes in murine dopaminergic neurotoxicity. Neurotoxicology，2018， 67：112-120.
, 百拇医药
[23] De Vivo DC， Trifiletti RR， Jacobson RI， Ronen GM， Behmand RA， Harik SI. Defective glucose transport across the blood-brain barrier as a cause of persistent hypoglycorrhachia， seizures， and developmental delay. N Engl J Med，1991， 325(10)：703-709.
[24] Pascual JM， Van Heertum RL， Wang D， Engelstad K， De Vivo DC. Imaging the metabolic footprint of Glut1 deficiency on the brain. Ann Neurol，2002， 52(4)：458-464., http://www.100md.com(周佳秀?田绍文?旷昕)
[13] van Eijsden P， Notenboom RG， Wu O， de Graan PN， van Nieuwenhuizen O， Nicolay K， Braun KP. In vivo 1H magnetic resonance spectroscopy， T2-weighted and diffusion-weighted MRI during lithium-pilocarpine-induced status epilepticus in the rat. Brain Res，2004， 1030(1)：11-18.
, http://www.100md.com
[14] Zsurka G， Kunz WS. Mitochondrial dysfunction and seizures： the neuronal energy crisis. Lancet Neurol，2015， 14(9)：956-966.
[15] Sada N， Lee S， Katsu T， Otsuki T， Inoue T. Epilepsy treatment. Targeting LDH enzymes with a stiripentol analog to treat epi-lepsy. Science，2015， 347(6228)：1362-1367.
[16] 翟瓊香，桂娟，乔惠宪，刘纪清. 小儿癫痫治疗前后脑细胞葡萄糖代谢改变及临床价值. 中山大学学报(医学科学版)，2008，29(2)：198-201.
[17] Shao LR， Stafstrom CE. Glycolytic inhibition by 2-deoxy-d-glu-cose abolishes both neuronal and network bursts in an in vitro seizure model. J Neurophysiol，2017， 118(1)：103-113.
, 百拇医药
[18] Oz G， DiNuzzo M， Kumar A， Moheet A， Seaquist ER. Revisiting glycogen content in the human brain. Neurochem Res，2015， 40(12)：2473-2481.
[19] Walls AB， Sickmann HM， Brown A， Bouman SD， Ransom B， Schousboe A， Waagepetersen HS. Characterization of 1，4-dideoxy-1，4-imino-d-arabinitol (DAB) as an inhibitor of brain glycogen shunt activity. J Neurochem，2008， 105(4)：1462-1470.
[20] DiNuzzo M， Mangia S， Maraviglia B， Giove F. Does abnormal glycogen structure contribute to increased susceptibility to seiz-ures in epilepsy？ Metab Brain Dis，2015， 30(1)：307-316.
, http://www.100md.com
[21] Gibbs ME， Lloyd HG， Santa T， Hertz L. Glycogen is a preferred glutamate precursor during learning in 1-day-old chick： biochemical and behavioral evidence. J Neurosci Res，2007， 85(15)：3326-3333.
[22] Johnson J Jr， Pajarillo E， Karki P， Kim J， Son DS， Aschner M， Lee E. Valproic acid attenuates manganese-induced reduction in expression of GLT-1 and GLAST with concomitant changes in murine dopaminergic neurotoxicity. Neurotoxicology，2018， 67：112-120.
, 百拇医药
[23] De Vivo DC， Trifiletti RR， Jacobson RI， Ronen GM， Behmand RA， Harik SI. Defective glucose transport across the blood-brain barrier as a cause of persistent hypoglycorrhachia， seizures， and developmental delay. N Engl J Med，1991， 325(10)：703-709.
[24] Pascual JM， Van Heertum RL， Wang D， Engelstad K， De Vivo DC. Imaging the metabolic footprint of Glut1 deficiency on the brain. Ann Neurol，2002， 52(4)：458-464., http://www.100md.com(周佳秀?田绍文?旷昕)