当前位置: 首页 > 期刊 > 《药物与人·相约健康》 > 2014年第8期 > 正文
编号:12712989
浅谈MircoRNA与糖尿病并发症(2)
http://www.100md.com 2014年8月1日 《药物与人》2014年第8期
     参考文献

    [1]Soifer, H.S., J.J. Rossi, and P. Saetrom, MicroRNAs in disease and potential therapeutic applications. Mol Ther, 2007. 15(12): p. 2070-9.

    [2]Tang, J. and T.S. Kern, Inflammation in diabetic retinopathy. Prog Retin Eye Res, 2011. 30(5): p. 343-58.

    [3]Kovacs, B., et al., MicroRNAs in early diabetic retinopathy in streptozotocin-induced diabetic rats. Invest Ophthalmol Vis Sci, 2011. 52(7): p. 4402-9.

    [4]Gregory, P.A., et al., The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1. Nat Cell Biol, 2008. 10(5): p. 593-601.

    [5]Chen, W.J., et al., The magic and mystery of microRNA-27 in atherosclerosis. Atherosclerosis, 2012. 222(2): p. 314-23.

    [6]Schilling, J.D., et al., Macrophages modulate cardiac function in lipotoxic cardiomyopathy. Am J Physiol Heart Circ Physiol, 2012. 303(11): p. H1366-73.

    [7]Miki, T., et al., Diabetic cardiomyopathy: pathophysiology and clinical features. Heart Fail Rev, 2013. 18(2): p. 149-66.

    [8]Cheng, Y. and C. Zhang, MicroRNA-21 in cardiovascular disease. J Cardiovasc Transl Res, 2010. 3(3):p. 251-5.

    [9]van Rooij, E., et al., Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis. Proc Natl Acad Sci U S A, 2008. 105(35): p. 13027-32. (邓晓威 谢宁)
上一页1 2