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过氧化氢通过激活氧化应激诱导成骨细胞凋亡(2)
http://www.100md.com 2013年11月1日 刘红等
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    参见附件。

     [3] Banfi G, Iorio EL, Corsi MM. Oxidative stress, free radicals and bone remodeling. Clin Chem Lab Med. 2008;46(11):1550-1555.

    [4] Reeve J, Hesp R, Wootton R. A new tracer method for the calculation of rates of bone formation and breakdown in osteoporosis and other generalised skeletal disorders. Calcif Tissue Res. 1976;22(2):191-206.

    [5] Weinstein RS, Manolagas SC. Apoptosis and osteoporosis. Am J Med. 2000;108(2):153-164.

    [6] Drummond GR, Selemidis S, Griendling KK, Sobey CG. Combating oxidative stress in vascular disease: NADPH oxidases as therapeutic targets. Nat Rev Drug Discov. 2011;10(6):453-471.

    [7] Marzani B, Felzani G, Bellomo RG, et al. Human muscle aging: ROS-mediated alterations in rectus abdominis and vastus lateralis muscles. Exp Gerontol. 2005;40(12):959-965.

    [8] Skulachev MV, Antonenko YN, Anisimov VN, et al. Mitochondrial-targeted plastoquinone derivatives. Effect on senescence and acute age-related pathologies. Curr Drug Targets. 2011;12(6):800-826.

    [9] Manolides AS, Cullen DM, Akhter MP. Effects of glucocorticoid treatment on bone strength. J Bone Miner Metab. 2010;28(5):532-539.

    

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