糖尿病周围神经病变发病机制研究(5)
[15] Shah GN， Morofuji Y， Banks WA， et al. High glucose-induced mitochondrial respiration and reactive oxygen species in mouse cerebral pericytes is reversed by pharmacological inhibition of mitochondrial carbonic anhydrases： Implications for cerebralmicrovascular disease in diabetes[J]. Biochem Biophys Res Commun， 2013， 440(2)： 354-358.
[16] Cipollone F， Iezzi A， Fazia M， et al. The receptor RAGE as a progression factor amplifying arachidonatedependent inflammatory and proteolyticresponse in human atherosclerotic plaques： role of glycemic control[J]. Circulation， 2003， 108(9)： 1070-1077.
[17] Lee JS， Lee Ys， Jeon B， et al. EC-SOD induces apoptosis through COX-2 and galectin-7 in the epidermis[J]. J Dermatol Sci， 2012， 65(2)： 126-133.
[18] Doupis J， Lyons TE， Wu S， et al. Microvascular reactivity and inflammatory cytokines in painful and painless peripheral diabetic neuropathy[J]. J Clin Endocrinol Metab， 2009， 94(6)： 2157-2163.
(收稿日期：2015-11-09), 百拇医药(施晓红)
[16] Cipollone F， Iezzi A， Fazia M， et al. The receptor RAGE as a progression factor amplifying arachidonatedependent inflammatory and proteolyticresponse in human atherosclerotic plaques： role of glycemic control[J]. Circulation， 2003， 108(9)： 1070-1077.
[17] Lee JS， Lee Ys， Jeon B， et al. EC-SOD induces apoptosis through COX-2 and galectin-7 in the epidermis[J]. J Dermatol Sci， 2012， 65(2)： 126-133.
[18] Doupis J， Lyons TE， Wu S， et al. Microvascular reactivity and inflammatory cytokines in painful and painless peripheral diabetic neuropathy[J]. J Clin Endocrinol Metab， 2009， 94(6)： 2157-2163.
(收稿日期：2015-11-09), 百拇医药(施晓红)