姜黄素对Aβ诱导的AD大鼠β—APP和BACE1基因表达的影响(2)
参考文献：
[1]Ono K，Naiki H，YamadaM，et al.The devolepment of preventives and therapeutics for Alzheimer′s disease that inhibite the for mation of beta-amyloid fibrils(fAbeta)，as well as destabize preformed fAbta[J].Curr Pham Des，2006，12(33)：4357-4375.
[2]尹红蕾，秦新月，王运良，等.姜黄素对海马内注射Aβ1-40后大鼠认知功能障碍的影响[J].中国实用神经疾病杂志，2009，12(9)：6-8.
[3]诸葛启钏，大鼠脑立体定位图谱[M].版.北京：人民卫生出版社，2005.
[4]Blennow K，de Leon MJ，Zetterberg H.Alzheimer's disease[J].Lancet，2006，368：387-403.
[5]Ohno M，Sametsky EA，Younkin LH et al.BACE1 deficiency rescues memorydeficits and cholinergic dysfunction in a mouse model of Alzheimer's disease[J].Neuron，2004，41：27-33.
[6]Chiocco MJ，Lamb BT.Spatial and temporal control of age-related APP processingin genomic-based beta-secretase transgenic mice[J].Neurobiol Aging，2007，28：75-84.
[7]Müller T，Meyer H E，Egensperger R，et al.The amyloid precursor proteinintracellular domain(AICD)as modulator of gene expression，apoptosis，andcytoskeletaldynamics-Relevance for Alzheimer's disease[J].Prog Neurobiol，2008，85：393-406.
[8]Volbracht C，Penzkofer S，Mansson D，et al.Measurement of cellular b-site of APPcleaving enzyme 1 activity and its modulation in neuronal assay systems[J].AnalBiochem，2009，387：208-220.
[9]Hardy J，Selkoe DJ.The amyloid hypothesis of Alzheimer's disease：progress andproblems on the road to therapeutics[J].Science，2002，297：353-356.编辑/王敏, 百拇医药(韩玉生等)
[1]Ono K，Naiki H，YamadaM，et al.The devolepment of preventives and therapeutics for Alzheimer′s disease that inhibite the for mation of beta-amyloid fibrils(fAbeta)，as well as destabize preformed fAbta[J].Curr Pham Des，2006，12(33)：4357-4375.
[2]尹红蕾，秦新月，王运良，等.姜黄素对海马内注射Aβ1-40后大鼠认知功能障碍的影响[J].中国实用神经疾病杂志，2009，12(9)：6-8.
[3]诸葛启钏，大鼠脑立体定位图谱[M].版.北京：人民卫生出版社，2005.
[4]Blennow K，de Leon MJ，Zetterberg H.Alzheimer's disease[J].Lancet，2006，368：387-403.
[5]Ohno M，Sametsky EA，Younkin LH et al.BACE1 deficiency rescues memorydeficits and cholinergic dysfunction in a mouse model of Alzheimer's disease[J].Neuron，2004，41：27-33.
[6]Chiocco MJ，Lamb BT.Spatial and temporal control of age-related APP processingin genomic-based beta-secretase transgenic mice[J].Neurobiol Aging，2007，28：75-84.
[7]Müller T，Meyer H E，Egensperger R，et al.The amyloid precursor proteinintracellular domain(AICD)as modulator of gene expression，apoptosis，andcytoskeletaldynamics-Relevance for Alzheimer's disease[J].Prog Neurobiol，2008，85：393-406.
[8]Volbracht C，Penzkofer S，Mansson D，et al.Measurement of cellular b-site of APPcleaving enzyme 1 activity and its modulation in neuronal assay systems[J].AnalBiochem，2009，387：208-220.
[9]Hardy J，Selkoe DJ.The amyloid hypothesis of Alzheimer's disease：progress andproblems on the road to therapeutics[J].Science，2002，297：353-356.编辑/王敏, 百拇医药(韩玉生等)