奥曲肽治疗先天性高胰岛素血症患儿的护理(3)
参考文献：
[1]Stanley CA. Hyperinsulinism in infants and children[J].Pediatr Clin North Am，1997， 44(2)： 363-374.
[2]Diva D De Leon，Stanley CA. Mechanism of Disease： advances in diagnosis and treatment of hyperinsulinism in neonates[J].Nature Clinical Practice Endocrinology and Metabolism，2007，3(1)：57-68.
[3]Flanagan Sarah E， Kapoor Ritika R， Hussain Khalid.Genetics of congenital hyperinsulinemic hypoglycemia[J].Seminars in Pediatric Surgery， 2011， 20(1)：13-17.
[4]Gong CX， Huang S， Su C， et al. Congenital hyperinsulinism in Chinese patients： 5-yr treatment outcome of 95 clinical cases with genetic analysis of 55 cases[J].Pediatric Diabetes，2016，17(3)：227-234.
[5]苏畅，巩纯秀.新生儿先天性高胰岛素血症15例临床分析[J].中国实用儿科杂志，2010，25(12)：940-942.
[6]Arnoux JB， de Lonlay P， Ribeiro MJ， et al. Congenital hyperinsulinism[J]. Early Human Development，2010，86(5)： 287-294.
[7]Molven A， Matre GE， Duran M， et al. Famillial hyperinsulinemic hypoglycemia caused by a defect in the SCHAD enzyme of mitochondrial fatty acid oxidation[J].Diabetes，2004，53(1)：221-227.
[8]Meissner T， Wendel U， Burqard P， et al. Long-term follow-up of 114 patients with congenital hyperinsulinism[J].European Journal of Endocrinology，2003，149(1)：43-51.
[9]Menni F， de Lonlay P， Sevin C， et al. Neurologic outcomes of 90 neonates and infants with persistent hyperinsulinemic hypoglycemia[J].Pediatrics，2001，107(3)： 476-479.
[10]Gataullina S， Dellatolas G， Perdry H， et al. Comorbidity and metabolic context are crucial factors determining neurological sequelae of hypoglycaemia[J].Developmental Medicine and Child Neurology，2012，54(11)： 1012-1017.
編辑/周芸霏, http://www.100md.com(李凤婷 王锐)
[1]Stanley CA. Hyperinsulinism in infants and children[J].Pediatr Clin North Am，1997， 44(2)： 363-374.
[2]Diva D De Leon，Stanley CA. Mechanism of Disease： advances in diagnosis and treatment of hyperinsulinism in neonates[J].Nature Clinical Practice Endocrinology and Metabolism，2007，3(1)：57-68.
[3]Flanagan Sarah E， Kapoor Ritika R， Hussain Khalid.Genetics of congenital hyperinsulinemic hypoglycemia[J].Seminars in Pediatric Surgery， 2011， 20(1)：13-17.
[4]Gong CX， Huang S， Su C， et al. Congenital hyperinsulinism in Chinese patients： 5-yr treatment outcome of 95 clinical cases with genetic analysis of 55 cases[J].Pediatric Diabetes，2016，17(3)：227-234.
[5]苏畅，巩纯秀.新生儿先天性高胰岛素血症15例临床分析[J].中国实用儿科杂志，2010，25(12)：940-942.
[6]Arnoux JB， de Lonlay P， Ribeiro MJ， et al. Congenital hyperinsulinism[J]. Early Human Development，2010，86(5)： 287-294.
[7]Molven A， Matre GE， Duran M， et al. Famillial hyperinsulinemic hypoglycemia caused by a defect in the SCHAD enzyme of mitochondrial fatty acid oxidation[J].Diabetes，2004，53(1)：221-227.
[8]Meissner T， Wendel U， Burqard P， et al. Long-term follow-up of 114 patients with congenital hyperinsulinism[J].European Journal of Endocrinology，2003，149(1)：43-51.
[9]Menni F， de Lonlay P， Sevin C， et al. Neurologic outcomes of 90 neonates and infants with persistent hyperinsulinemic hypoglycemia[J].Pediatrics，2001，107(3)： 476-479.
[10]Gataullina S， Dellatolas G， Perdry H， et al. Comorbidity and metabolic context are crucial factors determining neurological sequelae of hypoglycaemia[J].Developmental Medicine and Child Neurology，2012，54(11)： 1012-1017.
編辑/周芸霏, http://www.100md.com(李凤婷 王锐)