当前位置: 首页 > 期刊 > 《医学信息》 > 2019年第22期
编号:13450769
足细胞上皮间质转分化研究(4)
http://www.100md.com 2019年6月4日 《医学信息》 2019年第22期
     [19]Gil D,Ciolczyk-Wierzbicka D,Dulinska-Litewka J,et al.Themechanism of contribution of integrin linked kinase (ILK) to epithelial-mesenchymal transition (EMT)[J].Adv Enzyme Regul,2011,51(1):195-207.

    [20]Lin CL,Wang JY,et al.Wnt/β-catenin signaling modulates survival of high glucose-stressed mesangial cells[J].Journal of the American.society of Nephrology,2006,17(10):2812-2820.

    [21]Wu X,Gao Y,Xu L,et al.Exosomes from high glucose-treated glomerular endothelial cells trigger the epithelial-mesenchymal transition and dysfunction of podocytes[J].Sci Rep,2017,7(1):9371.

    [22]Zhou T,He X,Cheng R,et al.Implication of dysregulation of the canonical wingless-type MMTV integration site (WNT) pathway in diabetic nephropathy[J].Diabetologia,2012,55(1):255-266.

    [23]Feng Y,Ren J,Gui Y,et al.Wnt/β-Catenin-Promoted Macrophage Alternative Activation Contributes to Kidney Fibrosis[J].J Am Soc Nephrol,2018,29(1):182-193.

    [24]Shi G,Wu W,Wan YG,et al.Low dose of triptolide ameliorates podocyte epithelial-mesenchymal transition induced by high dose of D-glucose via inhibiting Wnt3α/β-catenin signaling pathway activation[J].China Journal of Chinese Materia Medica,2018,43(1):139-146.

    [25]Dai C,Stolz DB,et al.Wnt/beta-catenin signaling promotes podocyte dysfunction and albuminuria[J].J Am Soc Nephrol,2009,20(9):1997-2008.

    收稿日期:2019-9-11;修回日期:2019-9-23

    編辑/成森, http://www.100md.com(崔方强)
上一页1 2 3 4