当前位置: 首页 > 期刊 > 《中国社区医师》 > 20141
编号:13705206
MLK3与乳腺癌的关系(2)
http://www.100md.com 2014年1月8日 《中国社区医师》 20141
     参考文献

    1 Zhang H,Gallo KA.Auto inhibition of mixed lineage kinase 3through its SrcHomology 3 domain[J].BiolChem,2001,276(49):45598-45603.

    2 Nagai H,Noguchi T,Takeda K,et al.Pathophysiological roles of ASK1-MAP kinase signaling pathways[J].J Biochem Mol Biol,2007,40(1):1-6.

    3 Ip YT,Davis RJ.Signal transduction by the c-Jun N-terminal kinase(JNK)-from in Flammarion to development[J].Curropin Cell Bio,1998,10(2):205-219.

    4 Wang XT,Pei DS,Xu J,et al.Opposing effects of bad phosphorylation at two distinct sites by MLK1 and MLK3 on ischemic brain injury[J].Cell Signal,2007,19(9):1844-1856.

    5 Chen J,Li C,PeiDS,et al.GluR6-Containing KA Receptor Mediates the Activation of p38 MAP Kinase in Rat Hippocampal CA1 Region During Brain Ischemia Injury[J].Hippocampus,2009,19(1):79-89.

    6 Winn RA,Marek L,Han SY,et al.Restoration of Wnt-7a expression reverses non-small cell lung cancer cellular transformation through frizzled-9-mediated growth inhibition and promotion of cell differentiation[J].BioChem,2010,280(20):19625-19634.

    7 吴向华,陆云飞,黄名威.ERK及JNK/MAPK通路在复发转移乳腺癌中的表达及临床意义[J].广东医学,2010,31(14):1841-1843., http://www.100md.com(衣世明 王培林 冯晓东)
上一页1 2