细胞因子和抑郁症(4)
[19] Simmonsa D A, Broderick P A. Cytokines, stressors, and clinical depression: Augmented adaptation responses underlie depression pathogenesis. Progress in Neuro- Psychopharmacology & Biological Psychiatry, 2005, 29: 793 ~807
[20] 赵巍峰，刘少华，李莹莹等. 白介素-1β对大鼠自主行为影响的研究. 汕头大学医学院学报，2005，18(3)：141~143
[21] Broderic P A. Interleukin 1alpha alters hippocampal serotonin and norepinephrine release during open-field behavior in Sprague–Dawley animals: differences from the Fawn-Hooded animal model of depression. Progress In Neuro-Psychopharmacology & Biological Psychiatry, 2002, 26 (7–8): 1355~ 1372.
, 百拇医药
[22] Adrian J, Dunn, & Swiergiel A H. Effects of interleukin-1 and endotoxin in the forced swim and tail suspension tests in mice. Pharmacology, Biochemistry and Behavior, 2005, 81: 688 ~ 693
[23].Dunn A J, Swiergiel A H, Beaurepaire R. Cytokines as mediators of depression: what can we learn from animal studies? Neuroscience & Biobehavioral Reviews, 2005, 29: 891~909
[24] Charles L, Raison, Lucile C, et al. Cytokines sing the blues: inflammation and the pathogenesis of depression. Trends in Immunology, 2006, 27(1): 24~31
, 百拇医药
[25] Capuron L, Ravaud A, Neveu P J, et al. Association between decreased serum tryptophan concentrations and depressive symptoms in cancer patients undergoing cytokine therapy. Mol. Psychiatry, 2002, 7(5): 468~473
[26] 黄庆军，郝新玲. 脑内白介素-1β介导利血平引起的大鼠行为性抑郁. 中国行为医学科学，2003，12(5)：491~492
[27] Lestage D, Verrier K, Palin， et al. The enzyme indoleamine 2,3-dioxygenase is induced in the mouse brain in response to peripheral administration of lipopolysaccharide and superantigen. Brain, Behavior and Immunity, 2002, 16: 596~601
, http://www.100md.com
[28] David S, Glenn E F, Jon E. Differential inhibition of stress-induced adreocortical responses by 5-HT1A agonists and by 5-HT2 and 5-HT3 antagonists. Psychoneuroendocrinology, 1995, 20(3): 239~257
[29] Pariante C M, Pearce, B D, Pisell. T L, et al. The proinflammatory cytokine, interleukin-1a, reduces glucocorticoid receptor translocation and function. Endocrinology, 1999, 140: 4359~4366
[30] Wichers M C，Koek G H，Robaeys G，et al. IDO and interferon-alpha-induced depressive symptoms: a shift in hypothesis from tryptophan depletion to neurotoxicity. Mol Psychiatry, 2005, 10(6): 538~44
, http://www.100md.com
[31] Goehler L E, Relton J K, Dripps D, et al. Vagal paraganglia bind biotinylated interleukin-1 receptor antagonist: a possible mechanism for immune-to-brain communication. Brain Research Bulletin, 1997, 43(3): 357~364
[32] Deak T, Bellamy C, Agostino L J. Exposure to forced swim stress does not alter central production of IL-1. Brain Research, 2003, 972: 53~63
[ 上 页 ], http://www.100md.com(潘玉芹 林文娟)
[20] 赵巍峰，刘少华，李莹莹等. 白介素-1β对大鼠自主行为影响的研究. 汕头大学医学院学报，2005，18(3)：141~143
[21] Broderic P A. Interleukin 1alpha alters hippocampal serotonin and norepinephrine release during open-field behavior in Sprague–Dawley animals: differences from the Fawn-Hooded animal model of depression. Progress In Neuro-Psychopharmacology & Biological Psychiatry, 2002, 26 (7–8): 1355~ 1372.
, 百拇医药
[22] Adrian J, Dunn, & Swiergiel A H. Effects of interleukin-1 and endotoxin in the forced swim and tail suspension tests in mice. Pharmacology, Biochemistry and Behavior, 2005, 81: 688 ~ 693
[23].Dunn A J, Swiergiel A H, Beaurepaire R. Cytokines as mediators of depression: what can we learn from animal studies? Neuroscience & Biobehavioral Reviews, 2005, 29: 891~909
[24] Charles L, Raison, Lucile C, et al. Cytokines sing the blues: inflammation and the pathogenesis of depression. Trends in Immunology, 2006, 27(1): 24~31
, 百拇医药
[25] Capuron L, Ravaud A, Neveu P J, et al. Association between decreased serum tryptophan concentrations and depressive symptoms in cancer patients undergoing cytokine therapy. Mol. Psychiatry, 2002, 7(5): 468~473
[26] 黄庆军，郝新玲. 脑内白介素-1β介导利血平引起的大鼠行为性抑郁. 中国行为医学科学，2003，12(5)：491~492
[27] Lestage D, Verrier K, Palin， et al. The enzyme indoleamine 2,3-dioxygenase is induced in the mouse brain in response to peripheral administration of lipopolysaccharide and superantigen. Brain, Behavior and Immunity, 2002, 16: 596~601
, http://www.100md.com
[28] David S, Glenn E F, Jon E. Differential inhibition of stress-induced adreocortical responses by 5-HT1A agonists and by 5-HT2 and 5-HT3 antagonists. Psychoneuroendocrinology, 1995, 20(3): 239~257
[29] Pariante C M, Pearce, B D, Pisell. T L, et al. The proinflammatory cytokine, interleukin-1a, reduces glucocorticoid receptor translocation and function. Endocrinology, 1999, 140: 4359~4366
[30] Wichers M C，Koek G H，Robaeys G，et al. IDO and interferon-alpha-induced depressive symptoms: a shift in hypothesis from tryptophan depletion to neurotoxicity. Mol Psychiatry, 2005, 10(6): 538~44
, http://www.100md.com
[31] Goehler L E, Relton J K, Dripps D, et al. Vagal paraganglia bind biotinylated interleukin-1 receptor antagonist: a possible mechanism for immune-to-brain communication. Brain Research Bulletin, 1997, 43(3): 357~364
[32] Deak T, Bellamy C, Agostino L J. Exposure to forced swim stress does not alter central production of IL-1. Brain Research, 2003, 972: 53~63
[ 上 页 ], http://www.100md.com(潘玉芹 林文娟)