当前位置: 首页 > 期刊 > 《中外女性健康.下半月》 > 2014年第12期
编号:12803445
宫颈癌细胞表观遗传改变(2)
http://www.100md.com 2014年12月1日 中外女性健康·下半月 2014年第12期
     参考文献

    [1]朱景德.组学时代的表观遗传学研究的机遇和挑战[J].中国科学基金,2009,23(4):196-202.

    [2]Palefsky J.Biology of HPV in HIV infection[J].Adv Dent Res,2006,19(1):99-105.

    [3]Bhattacharjee B,Sengupta S.CpG methylation of HPV 16 LCR at e2 binding site proximal to P97 is associated with cervical cancer in presence of intact e2 [J].Virology, 2006,354(2):280-285.

    [4]Burgers WA, Blanchon L,Pradhan S, et al.Viral oncoproteins target the DnA methyltransferases [J].Oncogene,2007,26(11):1650-1655.

    [5]Anita Szalmás,et al. epigenetic alterations in cervical carcinogenesis[J]. Seminars in Cancer Biology,2009,19:144-152.

    [6]Henken, F.e, et al, Sequential gene promoter methylation during HPV-induced cervical carcinogenesis.Br J Cancer,2007.97(10):1457-64.

    [7]Zhang Z,et al.Retinoic acid receptor beta2 is epigenetically silenced either by DnA methylation or repressive histone modifications at the promoter in cervical cancer cells[J]. Cancer Lett,2007,318-27.

    [8]唐世倩,等.宫颈癌相关基因甲基化研究[J].临床医药实践,2009,18(3):163-165.

    [9]Hui-Juan Yang,et al,Differential DnA methylation profiles in gynecological cancers and correlation with clinico-pathological data. BMC CAnCeR,2006,6(19):212., 百拇医药(邢洁 朱笕青)
上一页1 2