伽玛射线对人垂体瘤细胞细胞周期和抑癌基因p16表达的影响(3)
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[2]Berckmans B, De Veylder L. Transcriptional control of the cell cycle [J]. Curr Opin Plant Biol,2009,12(5):599-605.
[3]Gravina GL, Festuccia C, Marampon F, et al. Biological rationale for the use of DNA methyltransferase inhibitors as new strategy for modulation of tumor response to chemotherapy and radiation [J]. Mol Cancer,2010,9(1):305.
[4]Teyssier F, Bay JO, Dionet C, et al. Cell cycle regulation after exposure to ionising radiation [J]. Bull Cancer,1999,86(4):345-347.
[5]Marretta RM, Ales F. Cancer cell(s) cycle sequencing reveals universal mechanisms of apoptosis [J]. Mol Cell Biomech,2010,7(4):225-266.
[6]Klassen NV, Walker PR, Ross CK, et al. Two-stage cell shrinkage and the OER for radiation-induced apoptosis of rat thymocytes[J]. Int J Radiat Biol,1993,64(5):571-581.
[7]Attri J, Srinivasan R, Majumdar S, et al. Alterations of tumor suppressor gene p16 INK 4a in pancreatic ductal carcinoma [J]. BMC Gastroenterol,2005,28(5):22-31.
[8]Liang X, Wang P, Gao Q, et al. Endogenous LKB1 knockdown accelerates G(1)/S transition through p53 and p16 pathways [J]. Cancer Biol Ther,2010,9(2):156-160.
[9]Machiavelli G, Cotignola J, Danilowicz K. Expression of p16(INK4A) gene in human pituitary tumours [J]. Pituitary,2008,11(1):71-75.
[10]Jaffrain-Rea ML, Ferretti E, Toniato E, et al. p16 (INK4a, MTS-1) gene polymorphism and methylation status in human pituitary tumours [J]. Clin Endocrinol (Oxf),1999,51(3):317-325.
(收稿日期：2011-01-14)
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