NF-κB对肝癌细胞自噬形成的影响(3)
[参考文献]
[1] 赵蔚然,许峰.NF-kB与肿瘤生成[J].西部医学,2010,22(9):1729-1731.
[2] Liu D,Liu XY,Robinson D,et al. Suppression of staphylococcal enterotoxin B-induced toxiticy by a import inhibitor [J]. J Biol Chem,2004,279(18):19239-19246.
[3] 邢飞跃,赵克森,姜勇.NF-κB的信号通路与阻断策略[J].中国病理生理杂志,2003,19(6):849-855.
[4] 姚辉华,王守立,朱宝松,等.NF-κB信号通路阻断对肝癌细胞SMMC7721增殖的影响[J]. 苏州大学学报:医学版,2009,29(1):38-40.
[5] 查勇,黄云超,寸英丽,等.NF-κB p65 siRNA对肝癌细胞株SM-7721凋亡和Bcl-2、Atg5、Beclin-1基因表达的影响[J].山东医药,2010,50(35):1-3.
[6] 叶青.自噬的分子机制与病理生理意义[J].国际病理科学与临床杂志,2007,27(8):358-362.
[7] 吕汕群,沈忠英.羊膜自噬的形态及其相关基因表达[J].中国医药导报,2008,5(22):11-13.
[8] Yong RJ,Narita M, Ferreira M,et al. Autophagy mediates the mitotin senescence transition [J]. Genes Dev,2009,23(7):798-803.
[9] White E,Dipaola RS. The double-edged sword of autophagy modulation in cancer [J]. Clinical Cancer Res,2009,15(17):5308-5316.
[10] He C,Klionsky DJ. Regulation mechanisms and signaling pathways of autophagy[J]. Annu Rev Genet,2009,43:67-93.
(收稿日期:2012-02-15 本文编辑:卫 轲), 百拇医药(郝高鹏 王世明 董秀山 任宁 范悦 卢玉东)
[1] 赵蔚然,许峰.NF-kB与肿瘤生成[J].西部医学,2010,22(9):1729-1731.
[2] Liu D,Liu XY,Robinson D,et al. Suppression of staphylococcal enterotoxin B-induced toxiticy by a import inhibitor [J]. J Biol Chem,2004,279(18):19239-19246.
[3] 邢飞跃,赵克森,姜勇.NF-κB的信号通路与阻断策略[J].中国病理生理杂志,2003,19(6):849-855.
[4] 姚辉华,王守立,朱宝松,等.NF-κB信号通路阻断对肝癌细胞SMMC7721增殖的影响[J]. 苏州大学学报:医学版,2009,29(1):38-40.
[5] 查勇,黄云超,寸英丽,等.NF-κB p65 siRNA对肝癌细胞株SM-7721凋亡和Bcl-2、Atg5、Beclin-1基因表达的影响[J].山东医药,2010,50(35):1-3.
[6] 叶青.自噬的分子机制与病理生理意义[J].国际病理科学与临床杂志,2007,27(8):358-362.
[7] 吕汕群,沈忠英.羊膜自噬的形态及其相关基因表达[J].中国医药导报,2008,5(22):11-13.
[8] Yong RJ,Narita M, Ferreira M,et al. Autophagy mediates the mitotin senescence transition [J]. Genes Dev,2009,23(7):798-803.
[9] White E,Dipaola RS. The double-edged sword of autophagy modulation in cancer [J]. Clinical Cancer Res,2009,15(17):5308-5316.
[10] He C,Klionsky DJ. Regulation mechanisms and signaling pathways of autophagy[J]. Annu Rev Genet,2009,43:67-93.
(收稿日期:2012-02-15 本文编辑:卫 轲), 百拇医药(郝高鹏 王世明 董秀山 任宁 范悦 卢玉东)