原发性高血压病患者血压水平与血浆抵抗素关系的研究(3)
[8] 李靖，赵杰娉，解金红，等.血清抵抗素在高血压子代人群中的变化.J Clin Cardiol(China)，2005，7:396-397.
[9] Mian ST， Shu YC， Dao MC， et al.Association of resistin gene 3’-untranslated region+62GA polymorphism with type 2 diabetes and hypertension in a chinese population.J Clin Endocrinol Meteb，2003，88:1258-1263.
[10] Kochan Z， Karbowska J.Dehydroepiandrosterone up-regulates resistin gene expression in white adipose tissue.Mol Cell Endocrinol，2004，218(1-2):57-64.
[11] 杨再刚，张木勋，张建华，等.蛋白激酶C对3T3-L1脂肪细胞抵抗素表达的影响.中国病理生理杂志。2006，22(8):1640-1643.
[12] Song H，Shojima N，Sakoda H，et al.Resistin is regulated by C/EBPα，PPARs，and signal-transducing molecules.Biochem Biophys Res Commun，2002，299(2):291-298.
[13] Tang X， Powelka AM， Soriano NA，et al.PTEN， but not SHIP2， suppresses insulin signaling through the phosphatidylinositol 3-kinase/Akt pathway in 3T3-L1 adipocytes.J Biol Chem,2005,280(23):22523-22529.
[14] Ono H， Katagiri H， Funaki M，et al.Regulation of phosphoinositide metabolism， Akt phosphorylation， and glucose transport by PTEN (phosphatase and tensin homolog deleted on chromosome 10) in 3T3-L1 adipocytes.Mol Endocrinol,2001,15(8):1411-1422.
[15] Ugi S，Imamura T，Maegawa H， et al.Protein phosphatase 2A negatively regulates insulin’s metabolic signaling pathway by inhibiting Akt (protein kinase B) activity in 3T3-L1 adipocytes.Mol Cell Biol,2004,24(19):8778-8789., 百拇医药(朱 云 杨再刚)
[9] Mian ST， Shu YC， Dao MC， et al.Association of resistin gene 3’-untranslated region+62GA polymorphism with type 2 diabetes and hypertension in a chinese population.J Clin Endocrinol Meteb，2003，88:1258-1263.
[10] Kochan Z， Karbowska J.Dehydroepiandrosterone up-regulates resistin gene expression in white adipose tissue.Mol Cell Endocrinol，2004，218(1-2):57-64.
[11] 杨再刚，张木勋，张建华，等.蛋白激酶C对3T3-L1脂肪细胞抵抗素表达的影响.中国病理生理杂志。2006，22(8):1640-1643.
[12] Song H，Shojima N，Sakoda H，et al.Resistin is regulated by C/EBPα，PPARs，and signal-transducing molecules.Biochem Biophys Res Commun，2002，299(2):291-298.
[13] Tang X， Powelka AM， Soriano NA，et al.PTEN， but not SHIP2， suppresses insulin signaling through the phosphatidylinositol 3-kinase/Akt pathway in 3T3-L1 adipocytes.J Biol Chem,2005,280(23):22523-22529.
[14] Ono H， Katagiri H， Funaki M，et al.Regulation of phosphoinositide metabolism， Akt phosphorylation， and glucose transport by PTEN (phosphatase and tensin homolog deleted on chromosome 10) in 3T3-L1 adipocytes.Mol Endocrinol,2001,15(8):1411-1422.
[15] Ugi S，Imamura T，Maegawa H， et al.Protein phosphatase 2A negatively regulates insulin’s metabolic signaling pathway by inhibiting Akt (protein kinase B) activity in 3T3-L1 adipocytes.Mol Cell Biol,2004,24(19):8778-8789., 百拇医药(朱 云 杨再刚)