自噬调节EMT过程在间质性肺病中的作用机制探讨(5)
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(收稿日期：2019-04-15), 百拇医药(徐存来 潘炯伟 金元虹)
[17] Mi S，Li Z，Yang HZ，et al. Blocking IL-17A promotes the resolution of pulmonary inflammation and fibrosis via TGF-beta1-dependent and -independent mechanisms[J]. J Immunol，2015，187(6)：3003-3014.
[18] Kuwano K，Araya J，Hara H，et al. Cellular senescence and autophagy in the pathogenesis of chronic obstructive pulmonary disease(COPD) and idiopathic pulmonary fibrosis(IPF)[J]. Respir Investig，2016，54(6)：397-406.
[19] Noble PW，Albera C，Bradford WZ，et al. Pirfenidone in patients with idiopathic pulmonary fibrosis (CAPACITY)：Two randomised trials[J]. Lancet，2014，377(9779)：1760-1769.
[20] Azuma A，TaguchiY，Ogura T，et al. Exploratory analysis of a phase III trial of pirfenidone identifies a subpopulation of patients with idiopathic pulmonary fibrosis as benefiting from treatment[J]. Respir Res，2014，12：143.
[21] Koesters R，Kaissling B，LehirM，et al. Tubular overexpression of transforming growth factor-beta1 induces autophagy and fibrosis but not mesenchymal transition of renal epithelial cells[J]. Am J Pathol，2014，177(2)：632-643.
(收稿日期：2019-04-15), 百拇医药(徐存来 潘炯伟 金元虹)