脓毒症休克心肌顿抑严重程度判断的临床研究(3)
[4] Dellinger RP，Levy MM，Carlet JM，et al. Surviving Sepsis Campaign: international guidelines for management of severe sepsis and septic shock: 2008[J]. Crit Care Med，2008，36(1)：296-327.
[5] Waisbren BA. Bacteremia due to gram - negative bacilli ot hert han t he Salmonella : a clinical and therapeutic study[J]. AMA ArchIntern Med，1951，88(4)：467-488.
[6] Flierl MA，Rit tirsch D，Huber-Lang MS，et al. Molecular Events in the Cardiomyopathy of Sepsis[J]. Mol Med，2008，14(5-6)：327-336.
[7] Dhainaut JF，Huyghebaert MF，Monsallier JF，et al. Coronary hemodynamics and myocardial metabolism of lactate, free fatty acids, glucose, and ketones in patients with septic shock[J]. Circulation，1987，75(3)：533-541.
[8] Watts JA，Kline JA，Thornton LR，et al. Metabolic dysfunction and depletion of mitochondria in hearts of septic rats[J]. J Mol Cell Cardiol，2004，36(1)：141-150.
[9] Levy RJ，Vijayasarathy C，Raj NR，et al. Competitive and noncompetitive inhibition of myocardial cytochrome C oxidase in sepsis[J]. Shock，2004，21(2)：110-114.
[10] Massion PB，Feron O，Dessy C，et al. Nitric oxide and cardiac function: Ten years after, and continuing [J]. Circ Res，2003，93(5)：388-398.
[11] Brune B. The intimate relation between nitric oxide and superoxide in apoptosis and cell survival[J]. Antioxid. Redox. Signal，2005，7(3-4)：497-507.
[12] Aviram Hochstadt，Yuval Meroz，Giora Landesberg，et al. Myocardial Dysfunction in Severe Sepsis and Septic Shock: More Questions Than Answers[J]. Journal of Cardiothoracic and Vascular Anesthesia，Vol xx，No x (Month)，2011，25(3)：526-535.
[13] 杨乐，邹晓静，李树生，等. 脓毒症心肌抑制研究进展[J]. 内科急危重症杂志，2010，16(1)：133-135.
(收稿日期：2012-05-11 本文编辑：马 双), 百拇医药(陈艳丽 郭富桃)
[5] Waisbren BA. Bacteremia due to gram - negative bacilli ot hert han t he Salmonella : a clinical and therapeutic study[J]. AMA ArchIntern Med，1951，88(4)：467-488.
[6] Flierl MA，Rit tirsch D，Huber-Lang MS，et al. Molecular Events in the Cardiomyopathy of Sepsis[J]. Mol Med，2008，14(5-6)：327-336.
[7] Dhainaut JF，Huyghebaert MF，Monsallier JF，et al. Coronary hemodynamics and myocardial metabolism of lactate, free fatty acids, glucose, and ketones in patients with septic shock[J]. Circulation，1987，75(3)：533-541.
[8] Watts JA，Kline JA，Thornton LR，et al. Metabolic dysfunction and depletion of mitochondria in hearts of septic rats[J]. J Mol Cell Cardiol，2004，36(1)：141-150.
[9] Levy RJ，Vijayasarathy C，Raj NR，et al. Competitive and noncompetitive inhibition of myocardial cytochrome C oxidase in sepsis[J]. Shock，2004，21(2)：110-114.
[10] Massion PB，Feron O，Dessy C，et al. Nitric oxide and cardiac function: Ten years after, and continuing [J]. Circ Res，2003，93(5)：388-398.
[11] Brune B. The intimate relation between nitric oxide and superoxide in apoptosis and cell survival[J]. Antioxid. Redox. Signal，2005，7(3-4)：497-507.
[12] Aviram Hochstadt，Yuval Meroz，Giora Landesberg，et al. Myocardial Dysfunction in Severe Sepsis and Septic Shock: More Questions Than Answers[J]. Journal of Cardiothoracic and Vascular Anesthesia，Vol xx，No x (Month)，2011，25(3)：526-535.
[13] 杨乐，邹晓静，李树生，等. 脓毒症心肌抑制研究进展[J]. 内科急危重症杂志，2010，16(1)：133-135.
(收稿日期：2012-05-11 本文编辑：马 双), 百拇医药(陈艳丽 郭富桃)