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塞来昔布对急性白血病原代细胞凋亡的影响(3)
http://www.100md.com 2012年7月5日 《中国医学创新》 2012年第19期
     [8] Adams J M,Cory S.The Bcl-2 apoptotic switch in cancer development and therapy[J].Oncogene,2007,26(9):1324-1337.

    [9] Yo R J,Strasser A.The BCL-2 protein family:opposing activities that mediate cell death[J].Nat Rev Mol Cell Biol,2008,9(1):47-59.

    [10] Rudner J,Elsaesser S J,Jendrossek V.Anti-apoptotic Bcl-2 fails to form efficient complexes with pro-apoptotic Bak to protect from Celecoxib-induced apoptosis[J].Biochemical Pharmacology,2011,81(1):32-42.

    [11] Liu B,Shi Z L,Feng J,et al.Celecoxib,a cyclooxygenase-2 inhibitor,induces apoptosis in human osteosarcoma cell line MG-63 via down-regulation of PI3K/Akt[J].Cell Biol int,2008,32(5):494-501.

    [12] Pyrko P,Soriano N,Kardosh A,et al.Downregulation of survivin expression and concomitant induction of apoptosis by celecoxib and its non-cyclooxygenase-2 inhibitory analogdimethy-celecoxib (DMC),in tumor cells in vitro and in vivo[J].Mol Cancer,2006,5(19):1476-1478.

    [13] Jerome K R,Sloan D D,Aubert M,et al.Measurement of CTL-induced cytotoxicity: the caspase 3 assay[J].Apoptosis,2003,8(6):563-571.

    [14] Fujimura S,Suzumiya J,Yamada Y.Downregulation of Bcl-xL and activation of caspases during retinoic acid-induced apoptosis in an adult T-cell leukemia cell line[J].Hematol,2003,4(5):328-335.

    (收稿日期:2012-03-26) (本文编辑:连胜利), 百拇医药(王瑞丽 阮林海 葛晓华)
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