CD4+CD25+Foxp3+Tregs在自身免疫性甲状腺疾病中的作用研究(4)
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[12]买文丽,韩清娟,曹文轩,等.人源化NOD小鼠中CD8+CD25+Tregs的频率和功能研究[J].免疫学杂志,2016,32(1):7-12.
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[14]郭彬彬,张梦军,任小珊,等.NOD小鼠人源化后CD4+T和CD8+T细胞及其分泌IFN-γ与IL-17的频率变化及意义[J].免疫学杂志,2015,31(2):93-99.
[15]谭燕,王晨虹.糖皮质激素诱导的肿瘤坏死因子受体在Tregs介导下的免疫耐受作用的研究进展[J].国际妇产科学杂志,2016,43(3):282-286.
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[17] Przybyl L,Ibrahim T,Haase N,et al.Regulatory T cells ameliorate intrauterine growth retardation in a transgenic rat model for preeclampsia[J].Hypertension,2015,65(6):1298-1306.
[18] Saifi B,Aflatoonian R,Tajik N,et al.Tregulatory markers expression in unexplained recurrent spontaneous abortion[J].
J Matern Fetal Neonatal Med,2016,29(7):1175-1180.
[19]莫俊鑾,周继昌,张丽君,等.硒对特应性皮炎小鼠CD4+CD25+Foxp3+调节性T细胞的影响[J].中国热带医学,2016,16(8):743-747.
[20]方周宾,林兵英,伍昌林,等.ICOS+Treg与B10细胞在AD患者发病机制中的作用[J].中国热带医学,2015,15(4):412-414.
(收稿日期:2017-04-12) (本文编辑:程旭然), 百拇医药(黄广英 禤文婷 何灵杰)
[12]买文丽,韩清娟,曹文轩,等.人源化NOD小鼠中CD8+CD25+Tregs的频率和功能研究[J].免疫学杂志,2016,32(1):7-12.
[13] Elessawy B,Li X C.Type 1 diabetes and T regulatory cells[J].Pharmacol Res,2015,98:22-30.
[14]郭彬彬,张梦军,任小珊,等.NOD小鼠人源化后CD4+T和CD8+T细胞及其分泌IFN-γ与IL-17的频率变化及意义[J].免疫学杂志,2015,31(2):93-99.
[15]谭燕,王晨虹.糖皮质激素诱导的肿瘤坏死因子受体在Tregs介导下的免疫耐受作用的研究进展[J].国际妇产科学杂志,2016,43(3):282-286.
[16] Petrillo M G,Ronchetti S,Ricci E,et al.GITR+ regulatory T cells in the treatment of autoimmune diseases[J].Autoimmun Rev,2015,14(2):117-126.
[17] Przybyl L,Ibrahim T,Haase N,et al.Regulatory T cells ameliorate intrauterine growth retardation in a transgenic rat model for preeclampsia[J].Hypertension,2015,65(6):1298-1306.
[18] Saifi B,Aflatoonian R,Tajik N,et al.Tregulatory markers expression in unexplained recurrent spontaneous abortion[J].
J Matern Fetal Neonatal Med,2016,29(7):1175-1180.
[19]莫俊鑾,周继昌,张丽君,等.硒对特应性皮炎小鼠CD4+CD25+Foxp3+调节性T细胞的影响[J].中国热带医学,2016,16(8):743-747.
[20]方周宾,林兵英,伍昌林,等.ICOS+Treg与B10细胞在AD患者发病机制中的作用[J].中国热带医学,2015,15(4):412-414.
(收稿日期:2017-04-12) (本文编辑:程旭然), 百拇医药(黄广英 禤文婷 何灵杰)