丝裂原活化蛋白激酶与缺血性脑血管病的关系(4)
[18] Benakis C，Bonny C，Hirt L.JNK inhibition and inflammation after cerebral ischemia[J].Brain Behavior & Immunity，2010，24(5)：800-811.
[19] Inose Y， Kato Y， Kitagawa K， et al.Activated microglia in ischemic stroke penumbra upregulate MCP-1 and CCR2 expression in response to lysophosphatidylcholine derived from adjacent neurons and astrocytes[J].Neuropathology，2015，35(3)：209-223.
[20] Dan L，Lei T，Kawano H，et al.Regulation and role of ERK phosphorylation in glial cells following a nigrostriatal pathway injury[J].Brain Research，2016，1648(Pt A)：90-100.
[21] Zhang J，Barasch N，Li R C，et al.Inhibition of cytosolic phospholipase A(2) alpha protects against focal ischemic brain damage in mice[J].Brain Research，2012，1471(3)：129-137.
[22] Chan P H.Role of the p38 mitogen-activated protein kinase/cytosolic phospholipase A2 signaling pathway in blood-brain barrier disruption after focal cerebral ischemia and reperfusion[J].J Cereb Blood Flow Metab，2008，28(10)：1686-1696.
[23] Wang Z K，Liu F F，Wang Y，et al.Let-7a gene knockdown protects against cerebral ischemia/reperfusion injury[J].Neural Regeneration Research，2016，11(2)：262-269.
[24] Zhen Y，Ding C，Sun J，et al.Activation of the calcium-sensing receptor promotes apoptosis by modulating the JNK/p38 MAPK pathway in focal cerebral ischemia-reperfusion in mice[J].American Journal of Translational Research，2016，8(2)：911-921.
[25]王旋，姜翠敏.永久性大腦中动脉闭塞大鼠纹状体P-SAPK/JNK表达与神经元凋亡的相关性[J].国际脑血管病杂志，2011，19(9)：667-672.
[26]任素伟，陈翔.p-p38MAPK在A2AR敲除新生小鼠缺氧缺血脑区的表达及意义[C]//全国小儿脑瘫康复学术会议暨国际学术交流会议，2012：36-42.
[27] Su D，Riley J，Armstead W M，et al.Salvinorin A pretreatment preserves cerebrovascular autoregulation after brain hypoxic/ischemic injury via extracellular signal-regulated kinase/mitogen-activated protein kinase in piglets[J].Anesthesia & Analgesia，2012，114(1)：200-204., http://www.100md.com(王锐 胡风云 赵晓霞 蔚洪恩)
[19] Inose Y， Kato Y， Kitagawa K， et al.Activated microglia in ischemic stroke penumbra upregulate MCP-1 and CCR2 expression in response to lysophosphatidylcholine derived from adjacent neurons and astrocytes[J].Neuropathology，2015，35(3)：209-223.
[20] Dan L，Lei T，Kawano H，et al.Regulation and role of ERK phosphorylation in glial cells following a nigrostriatal pathway injury[J].Brain Research，2016，1648(Pt A)：90-100.
[21] Zhang J，Barasch N，Li R C，et al.Inhibition of cytosolic phospholipase A(2) alpha protects against focal ischemic brain damage in mice[J].Brain Research，2012，1471(3)：129-137.
[22] Chan P H.Role of the p38 mitogen-activated protein kinase/cytosolic phospholipase A2 signaling pathway in blood-brain barrier disruption after focal cerebral ischemia and reperfusion[J].J Cereb Blood Flow Metab，2008，28(10)：1686-1696.
[23] Wang Z K，Liu F F，Wang Y，et al.Let-7a gene knockdown protects against cerebral ischemia/reperfusion injury[J].Neural Regeneration Research，2016，11(2)：262-269.
[24] Zhen Y，Ding C，Sun J，et al.Activation of the calcium-sensing receptor promotes apoptosis by modulating the JNK/p38 MAPK pathway in focal cerebral ischemia-reperfusion in mice[J].American Journal of Translational Research，2016，8(2)：911-921.
[25]王旋，姜翠敏.永久性大腦中动脉闭塞大鼠纹状体P-SAPK/JNK表达与神经元凋亡的相关性[J].国际脑血管病杂志，2011，19(9)：667-672.
[26]任素伟，陈翔.p-p38MAPK在A2AR敲除新生小鼠缺氧缺血脑区的表达及意义[C]//全国小儿脑瘫康复学术会议暨国际学术交流会议，2012：36-42.
[27] Su D，Riley J，Armstead W M，et al.Salvinorin A pretreatment preserves cerebrovascular autoregulation after brain hypoxic/ischemic injury via extracellular signal-regulated kinase/mitogen-activated protein kinase in piglets[J].Anesthesia & Analgesia，2012，114(1)：200-204., http://www.100md.com(王锐 胡风云 赵晓霞 蔚洪恩)
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