传染性单核细胞增多症患儿血清IL—21的变化及意义(2)
第1页 |
参见附件。
[3] Monteleone G,Pallone F,Macdonald T T.Interleukin-21(IL-21)-mediated pathways in T cell-mediated disease[J].Cytokine Growth Factor Rev,2009,20(2):185-191.
[4] Skak K,Frederiksen K S,Lundsgaard D.Interleukin-21 activates human natural killer cells and modulates their surface receptor expression[J].Immunology,2008,123(4):575-583.
[5] Kom T,Bettelli E,Gao W.IL-21 initiates an alternative pathway to induce proinflammatory TH17 cells[J].Nature,2007,448(7152):484-488.
[6] Zhou L,Ivanov I I,Spolski R,et al.IL-6 programs T(H)-17 cell differentiation by promoting sequentiM engagement of the 11,21 and JL-23 pathways[J].Nat Immunol,2007,8(9):967-974.
[7] Itoh S,Ohno T,Kakizki S,et al.Epstein-Barr virus-positive T-cell lymphoma cells having chromosome 22q11.2 deletion:An autopsy report of digeorge syndrome[J].Hum pathol,2011,42(12):2037-2041.
[8]宋建新,朱红艳,甸自金.传染性单核细胞增多症患儿外周血Th17细胞表达的研究[J].国际检验医学杂志,2012,33(1):25-26.
(收稿日期:2013-02-04) (编辑:程旭然)
您现在查看是摘要介绍页,详见PDF附件。