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FOXO调控下的细胞自噬及其与疾病的关系(4)
http://www.100md.com 2017年7月23日 《健康周刊》 201729
     [23]Ning Y, Li Z, Qiu Z. FOXO1 silence aggravates oxidative stress-promoted apoptosis in cardiomyocyt-es by reducing autophagy[J]. The Journal of toxicol-ogical sciences. 2015,40(5):637-45.

    [24]Chi Y, Shi C, Zhao Y, Guo C. Forkhead box O (FOXO) 3 modulates hypoxia-induced autophagy thr-ough AMPK signalling pathway in cardiomyocytes[J]. Bioscience reports. 2016,36(3).

    [25]徐俊, 黄秀兰. SIRT1-FoxO-自噬通路研究进展[J]. 中国药理学通报. 2014(07):901-4.

    [26]Leong A, Berkowitz SA, Triant VA, Porneala B, He W, Atlas SJ, et al. Hypoglycemia in Diabetes Mellitus as a Coronary Artery Disease Risk Factor in Patients at Elevated Vascular Risk[J]. The Journal of clinical endocrinology and metabolism. 2016,101(2):659-68.

    [27]Garrido AM, Bennett M. Assessment and conse-quences of cell senescence in atherosclerosis[J]. Cur-rent opinion in lipidology. 2016,27(5):431-8.

    [28]Moruno F, Perez-Jimenez E, Knecht E. Regulatio-n of autophagy by glucose in Mammalian cells[J]. Cells. 2012,1(3):372-95.

    基金項目:

    温州医科大学本专科学生科研项目(wyx2017101045)

    通讯作者:

    丛维涛,研究员, 百拇医药(何升曲 陈根 朱坤选 连燚沛 丛维涛)
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