《实验医学杂志》.2005年.第8期
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- IL-7 receptor signaling is necessary for stage transition in adult B cell development through up-regulation of EBF
- Protection from angiotensin II–mediated vasculotoxic and hypertensive response in mice lacking PI3K
- Intestinal antiinflammatory effect of 5-aminosalicylic acid is dependent on peroxisome proliferator–activated receptor-
- Structural and kinetic basis for heightened immunogenicity of T cell vaccines
- Among B cell non-Hodgkin's lymphomas, MALT lymphomas express a unique antibody repertoire with frequent rheumatoid factor reactivity
- Boosting antitumor responses of T lymphocytes infiltrating human prostate cancers
- Essential role for CD103 in the T cell–mediated regulation of experimental colitis
- Innate immunity against Francisella tularensis is dependent on the ASC/caspase-1 axis
- Single cell analysis shows decreasing FoxP3 and TGF1 coexpressing CD4+CD25+ regulatory T cells during autoimmune diabetes
- Rapid mobilization of murine and human hematopoietic stem and progenitor cells with AMD3100, a CXCR4 antagonist
- Contribution of DNA polymerase to immunoglobulin gene hypermutation in the mouse
- Deletion of DOCK2, a regulator of the actin cytoskeleton in lymphocytes, suppresses cardiac allograft rejection
- Fat meets the cholinergic antiinflammatory pathway
- SOCS-1 regulates IL-15–driven homeostatic proliferation of antigen-naive CD8 T cells, limiting their autoimmune potential
- Endogenous human cytomegalovirus gB is presented efficiently by MHC class II molecules to CD4+ CTL
- Actin and agonist MHC–peptide complex–dependent T cell receptor microclusters as scaffolds for signaling
- CD4+CD25+ regulatory T cells inhibit natural killer cell functions in a transforming growth factor––dependent manner
- Nucleic acids of mammalian origin can act as endogenous ligands for Toll-like receptors and may promote systemic lupus erythematosus
- Notch signaling is an important regulator of type 2 immunity
- The Scurfy mutation of FoxP3 in the thymus stroma leads to defective thymopoiesis
- Nutritional stimulation of cholecystokinin receptors inhibits inflammation via the vagus nerve
- CD44 is a physiological E-selectin ligand on neutrophils
- An anti-CD45RO/RB monoclonal antibody modulates T cell responses via induction of apoptosis and generation of regulatory T cells
- Natural selection drives recurrent formation of activating killer cell immunoglobulin-like receptor and Ly49 from inhibitory homologues
- Neutrophils mediate immune modulation of dendritic cells through glycosylation-dependent interactions between Mac-1 and DC-SIGN
- T cell–induced hyaluronan production by epithelial cells regulates inflammation