《实验医学杂志》.2006年.第5期
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- The c-FLIP–NH2 terminus(p22-FLIP) induces NF-B activation
- RAR is critical for maintaining a balance between hematopoietic stem cell self-renewal and differentiation
- Lymphocyte recruitment into the aortic wall before and during development of atherosclerosis is partially L-selectin dependent
- Toll-like receptor agonists influence the magnitude and quality of memory T cell responses after prime-boost immunization in nonhuman primat
- Human mesenchymal stem cells exert potent antitumorigenic effects in a model of Kaposi's sarcoma
- Platelets secrete stromal cell–derived factor 1 and recruit bone marrow–derived progenitor cells to arterial thrombi in vivo
- Telomerase flies the coop: the telomerase RNA component as a viral-encoded oncogene
- Protective immune responses against West Nile virus are primed by distinct complement activation pathways
- A virus-encoded telomerase RNA promotes malignant T cell lymphomagenesis
- Reprogramming of CTLs into natural killer–like cells in celiac disease
- The impact of DM on MHC class II–restricted antigen presentation can be altered by manipulation of MHC–peptide kinetic stability
- Bax does not have to adopt its final form to drive T cell death
- NOD.c3c4 congenic mice develop autoimmune biliary disease that serologically and pathogenetically models human primary biliary cirrhosis
- Bax does not have to adopt its final form to drive T cell death
- Mechanisms imposing the V bias of V14 natural killer T cells and consequences for microbial glycolipid recognition
- A critical role for the host mediator macrophage migration inhibitory factor in the pathogenesis of malarial anemia
- Fas-positive T cells regulate the resolution of airway inflammation in a murine model of asthma
- Cholesterol deficiency in a mouse model of Smith-Lemli-Opitz syndrome reveals increased mast cell responsiveness
- Plasmacytoid dendritic cells inhibit pulmonary immunopathology and promote clearance of respiratory syncytial virus
- Maintenance of viral suppression in HIV-1–infected HLA-B57+ elite suppressors despite CTL escape mutations