《高血压学杂志》.2006年.第5期
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- Different Impact of the Metabolic Syndrome on Left Ventricular Structure and Function in Hypertensive Men and Women
- Amino-Terminal Pro-B-Type Natriuretic Peptide and B-Type Natriuretic Peptide
- Association of Hemoglobin Delivery With Left Ventricular Structure and Function in Hypertensive Patients
- Electrocardiographic QRS Duration and the Risk of Congestive Heart Failure
- Longitudinal Mitral Annulus Velocities Are Reduced in Hypertensive Subjects With or Without Left Ventricle Hypertrophy
- Long-Term Risk of Mortality Associated With Selective and Combined Elevation in Office, Home, and Ambulatory Blood Pressure
- New Mass Spectrometric Assay for Angiotensin-Converting Enzyme 2 Activity
- Telmisartan But Not Valsartan Increases Caloric Expenditure and Protects Against Weight Gain and Hepatic Steatosis
- Impairment of In Vitro and In Vivo Heart Function in Angiotensin-(1-7) Receptor Mas Knockout Mice
- Increased Sympathetic Nerve Activity Correlates With Neurovascular Compression at the Rostral Ventrolateral Medulla
- Folate Supplementation During Pregnancy Improves Offspring Cardiovascular Dysfunction Induced by Protein Restriction
- Abnormalities of Retinal Microvascular Structure and Risk of Mortality From Ischemic Heart Disease and Stroke
- Effects of Continuous Positive Airway Pressure Versus Supplemental Oxygen on 24-Hour Ambulatory Blood Pressure
- Effects of Aging and Hypertension on the Microcirculation
- Serum Uric Acid Is Associated With Microalbuminuria in Prehypertension
- Office and Ambulatory Blood Pressure Are Independently Associated With Albuminuria in Older Subjects With Type 2 Diabetes
- Emergence of Novel Genetic Effects on Blood Pressure and Hemodynamics in Adolescence
- Rho Kinase Polymorphism Influences Blood Pressure and Systemic Vascular Resistance in Human Twins
- Polymorphism in Endothelin-Related Genes Limits Exercise-Induced Decreases in Arterial Stiffness in Older Subjects
- Association of the Ghrelin Receptor Gene Region With Left Ventricular Hypertrophy in the General Population
- Mechanisms Underlying Afterload-Induced Exacerbation of Myocardial Infarct Size
- Nonproteolytic Activation of Prorenin Contributes to Development of Cardiac Fibrosis in Genetic Hypertension
- Development of Heart Failure in Chronic Hypertensive Dahl Rats
- Vascular Endothelial Growth Factor Blockade Promotes the Transition From Compensatory Cardiac Hypertrophy to Failure in Response to Pressure
- Short Sleep Duration as a Risk Factor for Hypertension