《基因进展》.2006年.第12期

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• Genomic instability due to V(D)J recombination-associated transposition
• The deficiency of Akt1 is sufficient to suppress tumor development in Pten+/– mice
• Regulation of transcription factor latency by receptor-activated proteolysis
• A glycolytic enzyme, enolase, is recruited as a cofactor of tRNA targeting toward mitochondria in Saccharomyces cerevisiae
• The Wilms’ tumor 1(WT1) gene(+KTS isoform) functions with a CTE to enhance translation from an unspliced RNA with a retained intron
• Histone modification-dependent and -independent pathways for recruitment of checkpoint protein Crb2 to double-strand breaks
• Distinct functions of homeodomain-containing and homeodomain-less isoforms encoded by homothorax
• A role for the catalytic ribonucleoprotein RNase P in RNA polymerase III transcription
• Jmjd2b antagonizes H3K9 trimethylation at pericentric heterochromatin in mammalian cells
• The myocardin family of transcriptional coactivators: versatile regulators of cell growth, migration, and myogenesis
• Leukemia and lymphoma: a cost of doing business for adaptive immunity
• Polycomb-group proteins repressthe floral activator AGL19 in the FLC-independent vernalization pathway
• Fibroblast growth factor signals regulate a wave of Hedgehog activation that is essential for coronary vascular development