《循环研究杂志》.2005年.第11期
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- Enhanced Store OverloadeCInduced Ca2+ Release and Channel Sensitivity to Luminal Ca2+ Activation Are Common Defects of RyR2 Mutations Linked
- Bone Morphogenetic Protein Signaling Modulates Myocardin Transactivation of Cardiac Genes
- FcRIIB Mediates C-Reactive Protein Inhibition of Endothelial NO Synthase
- Ca2+ InfluxeCInduced Sarcoplasmic Reticulum Ca2+ Overload Causes Mitochondrial-Dependent Apoptosis in Ventricular Myocytes
- Calreticulin Destabilizes Glucose Transporter-1 mRNA in Vascular Endothelial and Smooth Muscle Cells Under High-Glucose Conditions
- Haplotype Effect of the Matrix Metalloproteinase-1 Gene on Risk of Myocardial Infarction
- Discrepancies Between Nitroglycerin and NO-Releasing Drugs on Mitochondrial Oxygen Consumption, Vasoactivity, and the Release of NO
- NADPH Oxidase Activity and Function Are Profoundly Greater in Cerebral Versus Systemic Arteries
- Synthetic Retinoid Am80 Suppresses Smooth Muscle Phenotypic Modulation and In-Stent Neointima Formation by Inhibiting KLF5
- Nitric Oxide Regulates Transforming Growth Factor- Signaling in Endothelial Cells
- Nuclear RNA Foci in the Heart in Myotonic Dystrophy
- Platelet-Derived Growth Factor D Induces Cardiac Fibrosis and Proliferation of Vascular Smooth Muscle Cells in Heart-Specific Transgenic Mic
- Mobilization of Bone MarroweCDerived Cells Enhances the Angiogenic Response to Hypoxia Without Transdifferentiation Into Endothelial Cells
- Intrasarcoplasmic Amyloidosis Impairs Proteolytic Function of Proteasomes in Cardiomyocytes by Compromising Substrate Uptake
- Progesterone Antagonizes the Vasoprotective Effect of Estrogen on Antioxidant Enzyme Expression and Function
- Dual Pathways for Nuclear Factor B Activation by Angiotensin II in Vascular Smooth Muscle
- Embryonic even skippedeCDependent Muscle and Heart Cell Fates Are Required for Normal Adult Activity, Heart Function, and Lifespan
- Impaired CXCR4 Signaling Contributes to the Reduced Neovascularization Capacity of Endothelial Progenitor Cells From Patients With Coronary
- Cardiac Myosin-Binding Protein-C Phosphorylation and Cardiac Function
- Myocardin Enhances Smad3-Mediated Transforming Growth Factor-1 Signaling in a CArG Box-Independent Manner
- Activation of the Endothelial Store-Operated ISOC Ca2+ Channel Requires Interaction of Protein 4.1 With TRPC4
- Effects of D-4F on Vasodilation and Vessel Wall Thickness in Hypercholesterolemic LDL ReceptoreCNull and LDL Receptor/Apolipoprotein A-I Dou
- Ischemic Neoangiogenesis Enhanced by 2-Adrenergic Receptor Overexpression