《循环研究杂志》.2005年.第12期
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- Gab1, SHP2, and Protein Kinase A Are Crucial for the Activation of the Endothelial NO Synthase by Fluid Shear Stress
- Protective Effect of the KCNMB1 E65K Genetic Polymorphism Against Diastolic Hypertension in Aging Women and Its Relevance to Cardiovascular
- TRPV4 Forms a Novel Ca2+ Signaling Complex With Ryanodine Receptors and BKCa Channels
- Oxidative Mediated Lipid Peroxidation Recapitulates Proarrhythmic Effects on Cardiac Sodium Channels
- cAMP-Binding Protein Epac Induces Cardiomyocyte Hypertrophy
- ExcitationeCContraction Coupling in Na+eCCa2+ Exchanger Knockout Mice
- Contribution of Kv Channels to Phenotypic Remodeling of Human Uterine Artery Smooth Muscle Cells
- Phospholipase C Modulates -Adrenergic ReceptoreC Dependent Cardiac Contraction and Inhibits Cardiac Hypertrophy
- Ca2+/CalmodulineCDependent Protein Kinase Modulates Cardiac Ryanodine Receptor Phosphorylation and Sarcoplasmic Reticulum Ca2+ Leak in Heart
- Increased Neointima Formation in Cysteine-Rich Protein 2eCDeficient Mice in Response to Vascular Injury
- Targeted Deletion of Kv4.2 Eliminates Ito,f and Results in Electrical and Molecular Remodeling, With No Evidence of Ventricular Hypertrophy
- Nuclear Targeting of Akt Enhances Ventricular Function and Myocyte Contractility
- PTEN Activity Is Modulated During Ischemia and Reperfusion
- Soluble Vascular Endothelial Growth Factor Receptor-1(sFLT-1) Mediates Downregulation of FLT-1 and Prevents Activated Neutrophils From Wome
- Adiponectin Inhibits Endothelial Synthesis of Interleukin-8