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临床研究杂志
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2004年
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3期
: 共 22 篇
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3期
[
2005-5-5
]
Achieving antigen-specific immune regulation
[
2005-5-5
]
An inauspicious start for the US National Biospecimen Network
[
2005-5-5
]
Autoreactive T cell responses show proinflammatory polarization in diabetes but a regulatory phenotype in health
[
2005-5-5
]
Central role of mitochondrial aldehyde dehydrogenase and reactive oxygen species in nitroglycerin tolerance and cross-tolerance
[
2005-5-5
]
Central role of the P2Y12 receptor in platelet activation
[
2005-5-5
]
Conditional disruption of IB kinase 2 fails to prevent obesity-induced insulin resistance
[
2005-5-5
]
Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C–deficient mice
[
2005-5-5
]
Defects in secretion, aggregation, and thrombus formation in platelets from mice lacking Akt2
[
2005-5-5
]
ENU mutagenesis identifies mice with mitochondrial branched-chain aminotransferase deficiency resembling human maple syrup urine disease
[
2005-5-5
]
Extensive tissue-regenerative capacity of neonatal human keratinocyte stem cells and their progeny
[
2005-5-5
]
Glycoprotein 130 regulates bone turnover and bone size by distinct downstream signaling pathways
[
2005-5-5
]
Helicobacter pylori persistence: biology and disease
[
2005-5-5
]
High vaccination efficiency of low-affinity epitopes in antitumor immunotherapy
[
2005-5-5
]
How do mutations in lamins A and C cause disease?
[
2005-5-5
]
Lamin A/C deficiency causes defective nuclear mechanics and mechanotransduction
[
2005-5-5
]
Nitrate tolerance oxidative stress and mitochondrial function: another worrisome chapter on the effects of organic nitrates
[
2005-5-5
]
Salicylate induces an antibiotic efflux pump in Burkholderia cepacia complex genomovar III (B. cenocepacia)
[
2005-5-5
]
Site and mechanism of leptin action in a rodent form of congenital lipodystrophy
[
2005-5-5
]
Tandem mass spectrometry in discovery of disorders of the metabolome
[
2005-5-5
]
Tandem mass spectrometry in discovery of disorders of the metabolome
[
2005-5-5
]
The human societal and scientific legacy of cholera
[
2005-5-5
]
TNF- is a critical negative regulator of type 1 immune activation during intracellular bacterial infection